BMS11-1039 - Pharmacology of diuretics

Flashcards by Evian Chai, created more than 1 year ago

How thiazides and loop diuretics increase urine production [giving site of action/mechanism] and why they can contribute to hypokalaemia. How K+ sparing diuretics increase urine production, and why they can be used to prevent hypokalaemia. Where and how ACE inhibitors and Angiotensin II antagonists have their effect.

	Created by Evian Chai about 5 years ago

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What are diuretics?

What occurs in oedematous conditions? Which type of diuretic is used to treat it?

Which type of diuretic is used to treat hypertension? How does it work?

What are three types of oedematous conditions?

Where are 2/3 of H20/salts, all glucose, aa, and HCO3- reabsorbed?

Where is 25% of NaCl reabsored?

Where is 10% of Na+ reabsorbed?

In the collecting tubule what regulates Na+ reabsorption/ K+ secretion?

What part of the kidney does Loop diuretics work on?

What part of the kidney does Thiazide diuretics work on?

What part of the kidney does K+ Sparing diuretics work on?

For loop diuretics, what is its:
1. Mechanism
2. Used for
3. Side effects

For thiazide diuretics, what is its:
1. Mechanism
2. Used for
3. Side effects

For K+ spanning diuretics, what is its:
1. Mechanism
2. Used for
3. Side effects

What do aldosterone blockers (eg. Spironalactone) do?
Where?
What is it used for?

Why do loop diuretics and thiazide blockers cause hypokalaemia?

What is the most effective diuretic? Why?

Furesemide/Turasemide

Hydrocholorothiazide, Bendroflumethiazide

Amiloride, Triamterene